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Medical uses

Oxycodone is effective for managing moderate to moderately severe acute or chronic pain.[7] It has been found to improve quality of life for those with many types of pain.[8]

Oxycodone can also be used as a alternative to other opiates to treat severe diarrhea and diarrhea predominant irritable bowel syndrome when drugs such as loperamide and diphenoxylate are ineffective.

In 2001, the European Association for Palliative Care recommended that oral oxycodone be a second-line alternative to oral morphine for cancer pain.[9] There is no evidence that any opioids are superior to morphine in relieving the pain of cancer, and no controlled trials have shown oxycodone to be superior to morphine.[10]

[edit] Adverse effects

Main side effects of oxycodone[11]

The most commonly reported effects include memory loss, constipation, fatigue, dizziness, nausea, lightheadedness, headache, dry mouth, anxiety, pruritus, and diaphoresis.[12] It has also been claimed to cause dimness in vision due to miosis. Some patients have also experienced loss of appetite, nervousness, abdominal pain, diarrhea, ischuria, dyspnea, and hiccups,[2] although these symptoms appear in less than 5% of patients taking oxycodone. Rarely, the drug can cause impotence, enlarged prostate gland, and decreased testosterone secretion. Compared to morphine, oxycodone causes less respiratory depression, sedation, pruritus, and nausea.[13] As a result, it is generally better tolerated than morphine.[14]

In high doses, overdoses, or in patients not tolerant to opiates, oxycodone can cause shallow breathing, bradycardia, cold, clammy skin, apnea, hypotension, miosis (pupil constriction), circulatory collapse, respiratory arrest, and death.[2]

[edit] Withdrawal

There is a high risk of experiencing severe withdrawal symptoms if a patient discontinues oxycodone abruptly. Therefore therapy should be discontinued gradually rather than abruptly. People who use oxycodone in a hazardous or harmful fashion are at even higher risk of severe withdrawal symptoms as they tend to use higher than prescribed doses. The symptoms of oxycodone withdrawal are the same as for other opiate based painkillers and may include “anxiety, panic attack, nausea, insomnia, muscle pain, muscle weakness, fevers, and other flu like symptoms.”[15]

Withdrawal symptoms have also been reported in newborns whose mothers had been either injecting or orally taking oxycodone during pregnancy.[16]

[edit] Detection in biological fluids

Oxycodone and/or its major metabolites may be quantitated in blood or urine to monitor for abuse, confirm a diagnosis of poisoning or assist in a medicolegal death investigation. Many commercial opiate screening tests cross-react appreciably with oxycodone and its metabolites, but chromatographic techniques can easily distinguish oxycodone from other opiates.[17]

[edit] Pharmacology

[edit] Mechanism of action

A group of Australian researchers has proposed (based on a 1997 study in rats) that oxycodone, unlike morphine (the effect of which is mediated by μ-opioid receptors), acts on κ-opioid receptors.[18] Further research by this group indicates the drug appears to be a κ2b-opioid agonist.[19] However, this has been disputed, primarily on the basis that oxycodone produces effects typical of μ-opioid agonists.[20]

Research by a Japanese group suggests that the effect of oxycodone is mediated by different receptors in different situations. Specifically, in diabetic mice the κ-opioid receptor appears to be involved in the antinociceptive effects of oxycodone,[21] while in non-diabetic mice the μ1-opioid receptor seems to be primarily responsible for these effects.[22]

[edit] Absorption

After a dose of conventional oral oxycodone, peak plasma levels of the drug are attained in approximately one hour;[3] in contrast, after a dose of OxyContin (an oral continuous release formulation), peak plasma levels of oxycodone occur in about three hours.[2]

[edit] Distribution

Oxycodone in the blood is distributed to skeletal muscle, liver, intestinal tract, lungs, spleen, and brain.[2] Conventional oral preparations of oxycodone start to reduce pain within 10–15 minutes; in contrast, OxyContin starts to reduce pain within 1 hour.[7]

[edit] Metabolism

Oxycodone is metabolized to α and β oxycodol; oxymorphone, then α and β oxymorphol and noroxymorphone; and noroxycodone, then α and β noroxycodol and noroxymorphone (N-desmethyloxycodone).[3] (14-Hydroxydihydrocodeine that in turn becomes 14-Hydroxydihydromorphine) These metabolites are true only for humans.[23] As many as six metabolites for oxycodone (14-hydroxydihydromorphinone, 14-hydroxydihydrocodeine, 14-hydroxydihydrocodeinone N-oxide {oxycodone N-oxide}, 14-hydroxydihydroisocodeine, 14-hydroxydihydrocodeine N-oxide, and noroxycodone) have been found in rabbits,[24] several of which are thought to be active metabolites to some extent, although a study using conventional oral oxycodone concluded that oxycodone itself, and not its metabolites, is predominantly responsible for the drug’s opioid effects on the brain.[3]

Unlike morphine and hydromorphone, oxycodone is metabolized by the cytochrome P450 enzyme system in the liver, making it vulnerable to drug interactions.[2] Some people are fast metabolizers resulting in reduced analgesic effect but increased adverse effects, while others are slow metabolisers resulting in increased toxicity without improved analgesia.[25][26] The dose of OxyContin must be reduced in patients with reduced hepatic function.[7]

[edit] Elimination

Oxycodone and its metabolites are mainly excreted in the urine and sweat; therefore, it accumulates in patients with renal impairment.[7]

[edit] Dosage and administration

Oxycodone can be administered orally, intranasally, via intravenous/intramuscular/subcutaneous injection or rectally. The bioavailability of oral administration of Oxycodone averages 60–87%, with rectal administration yielding the same results; intranasal varies between individuals with a mean of 46%.[27]

Oxycodone is approximately 1.5–2 times as potent as morphine when administered orally.[28][29] However, 10–15 mg of oxycodone produces an analgesic effect similar to 10 mg of morphine when administered intramuscularly.[30] Therefore, as a parenteral dose, morphine is approximately up to 50% more potent than oxycodone.

There are no comparative trials showing that oxycodone is more effective than any other opioid. In palliative care, morphine remains the gold standard;[10] however, oxycodone can be useful as an alternative opioid if a patient has troublesome adverse effects with morphine.

[edit] Chemistry

Oxycodone’s chemical name is derived from codeine. The chemical structures are very similar, differing only in that

  • Oxycodone has a hydroxyl group at carbon-14 (codeine has just a hydrogen in its place), hence oxycodone;
  • Oxycodone has a 7,8-dihydro feature, whereas codeine has a double bond between those two carbons; and
  • Oxycodone has a carbonyl group (as in ketones) in place of the hydroxyl group of codeine, hence the “-one” suffix.

It is also similar to hydrocodone, differing only in that it has a hydroxyl group at carbon-14.[7]

Expanded expression for the compound oxycodone in the academic literature include “dihydrohydroxycodeinone”,[4][31][32] “Eucodal”,[31][32] “Eukodal”,[1][33] “14-hydroxydihydrocodeinone”,[4][31] and “Nucodan”.[31][32] In a UNESCO convention, the translations of “oxycodone” are oxycodon (Dutch), oxycodone (French), oxicodona (Spanish), الأوكسيكودون (Arabic), 羟考酮 (Chinese), and оксикодон (Russian).[34] The word “oxycodone” should not be confused with “oxandrolone“, “oxazepam“, “oxybutynin“, “oxytocin“, or “Roxanol“.[35]

 Brand names

All brand-names not marked otherwise are standard-release tablets or capsules of oxycodone without further pharmacologically active ingredients.

  • Dinarkon (Germany)
  • Diphydrone
  • Endocet (US) (compounded with paracetamol)—by Endo
  • Endodan (US) (compounded with acetylsalicylic acid)—by Endo
  • ETH-Oxydose (US)
  • Endone (US)—by Endo
  • Eukodal (Germany) (for injection)
  • OxyContin (US) (modified release)—by Purdue Frederick
  • OxyFast (US) (oral liquid)
  • OxyNorm (US) (tablets, oral liquid, liquid for injection)
  • OxyNormoro (France)—by Mundipharma
  • Pancodine
  • Percodan (US) (compounded with acetylsalicylic acid)—by Endo
  • Percodan-Demi (US) (compounded with acetylsalicylic acid)—by Endo
  • Percocet (US) (compounded with paracetamol)—by Endo
  • Percolone (US)—by Endo
  • Roxicet (US)—by Roxane
  • Roxicodone (US)—by Roxane
  • Roxilox (US)—by Roxane
  • Roxiprin (US) (compounded with acetylsalicylic acid)—by Roxane
  • Supeudol (Canada)—by Sandoz
  • Thekodin (Germany)
  • Tylox (US)

[edit] History

Freund and Speyer of the University of Frankfurt in Germany first synthesized oxycodone from thebaine in 1916,[6] a few years after the German pharmaceutical company Bayer had stopped the mass production of heroin due to hazardous use, harmful use, and dependence. It was hoped that a thebaine-derived drug would retain the analgesic effects of morphine and heroin with less dependence. To some extent this was achieved, as oxycodone does not have the same immediate effect as heroin or morphine nor does it last as long.

The first clinical use of the drug was documented in 1917, the year after it was first developed.[6][33] It was first introduced to the US market in May 1939. In early 1928, Merck introduced a combination product containing scopolamine, oxycodone, andephedrine under the German initials for the ingredients SEE, which was later renamed Scophedal (SCOpolamine ePHEDrine & eukodAL)—this combination is essentially an oxycodone analogue of the morphine-based Twilight Sleep with ephedrine added to reduce circulatory and respiratory effects.

The International Narcotics Control Board estimates that 11.5 tons (23,000 lbs) of oxycodone were manufactured worldwide in 1998,[36] by 2007 this figure had grown to 75.2 tons (150,400 lbs).[36] Of all countries, the United States had the highest total consumption of oxycodone in 2007 at 51.6 tons (103,200 lbs) or 82% of the world total. This translates into over half a billion 80 mg tablets per year.[36] In addition, the U.S. had the highest per capita consumption of oxycodone, followed by Canada in 2006, 2007 and 2008. This makes the consumption of oxycodone in North America (United States and Canada alone) higher than all other countries of the world combined. The United States and Canada are consistently ranked among the top two or three in most type of opioid consumption per capita (for example, in 2006 Canada ranked second and the U.S. ranked third in morphine consumption per capita; only Austria ranked higher and that is due to the fact that in addition to being used to treat moderate to severe pain, morphine is also used to treat heroin or morphine dependence in Austria – a practice which is unheard of in North America).[36][37]
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5 Responses to “Buy percocet online no prescription is ever required”

  1. purchasepercocetonline Says:

    Oxycodone oral medications are generally prescribed for the relief of moderate to severe pain. Currently it is formulated as single ingredient products or compounded products. Some common examples of compounding are oxycodone with acetaminophen/paracetamol or NSAIDs such as ibuprofen. The formulations are available as generics but are also made under various brand names. OxyContin is Purdue Pharma’s brand for time-release oral oxycodone. The manufacturing rights to time-released generic oxycodone are under dispute

  2. purchasepercocetonline Says:

    Oxycodone is effective for managing moderate to moderately severe acute or chronic pain.[7] It has been found to improve quality of life for those with many types of pain

  3. purchasepercocetonline Says:

    Oxycodone can also be used as a alternative to other opiates to treat severe diarrhea and diarrhea predominant irritable bowel syndrome when drugs such as loperamide and diphenoxylate are ineffective.

    In 2001, the European Association for Palliative Care recommended that oral oxycodone be a second-line alternative to oral morphine for cancer pain.[9] There is no evidence that any opioids are superior to morphine in relieving the pain of cancer, and no controlled trials have shown oxycodone to be superior to morphine.

  4. purchasepercocetonline Says:

    Medical usesOxycodone is effective for managing moderate to moderately severe acute or chronic pain.[7] It has been found to improve quality of life for those with many types of pain.[8]

    Oxycodone can also be used as a alternative to other opiates to treat severe diarrhea and diarrhea predominant irritable bowel syndrome when drugs such as loperamide and diphenoxylate are ineffective.

    In 2001, the European Association for Palliative Care recommended that oral oxycodone be a second-line alternative to oral morphine for cancer pain.[9] There is no evidence that any opioids are superior to morphine in relieving the pain of cancer, and no controlled trials have shown oxycodone to be superior to morphine.[10]

    [edit] Adverse effects
    Main side effects of oxycodone[11]The most commonly reported effects include memory loss, constipation, fatigue, dizziness, nausea, lightheadedness, headache, dry mouth, anxiety, pruritus, and diaphoresis.[12] It has also been claimed to cause dimness in vision due to miosis. Some patients have also experienced loss of appetite, nervousness, abdominal pain, diarrhea, ischuria, dyspnea, and hiccups,[2] although these symptoms appear in less than 5% of patients taking oxycodone. Rarely, the drug can cause impotence, enlarged prostate gland, and decreased testosterone secretion. Compared to morphine, oxycodone causes less respiratory depression, sedation, pruritus, and nausea.[13] As a result, it is generally better tolerated than morphine.[14]

    In high doses, overdoses, or in patients not tolerant to opiates, oxycodone can cause shallow breathing, bradycardia, cold, clammy skin, apnea, hypotension, miosis (pupil constriction), circulatory collapse, respiratory arrest, and death.[2]

    [edit] WithdrawalThere is a high risk of experiencing severe withdrawal symptoms if a patient discontinues oxycodone abruptly. Therefore therapy should be discontinued gradually rather than abruptly. People who use oxycodone in a hazardous or harmful fashion are at even higher risk of severe withdrawal symptoms as they tend to use higher than prescribed doses. The symptoms of oxycodone withdrawal are the same as for other opiate based painkillers and may include “anxiety, panic attack, nausea, insomnia, muscle pain, muscle weakness, fevers, and other flu like symptoms.”[15]

    Withdrawal symptoms have also been reported in newborns whose mothers had been either injecting or orally taking oxycodone during pregnancy.[16]

    [edit] Detection in biological fluidsOxycodone and/or its major metabolites may be quantitated in blood or urine to monitor for abuse, confirm a diagnosis of poisoning or assist in a medicolegal death investigation. Many commercial opiate screening tests cross-react appreciably with oxycodone and its metabolites, but chromatographic techniques can easily distinguish oxycodone from other opiates.[17]

    [edit] Pharmacology

  5. purchasepercocetonline Says:

    Pharmacology[edit] Mechanism of actionA group of Australian researchers has proposed (based on a 1997 study in rats) that oxycodone, unlike morphine (the effect of which is mediated by μ-opioid receptors), acts on κ-opioid receptors.[18] Further research by this group indicates the drug appears to be a κ2b-opioid agonist.[19] However, this has been disputed, primarily on the basis that oxycodone produces effects typical of μ-opioid agonists.[20]

    Research by a Japanese group suggests that the effect of oxycodone is mediated by different receptors in different situations. Specifically, in diabetic mice the κ-opioid receptor appears to be involved in the antinociceptive effects of oxycodone,[21] while in non-diabetic mice the μ1-opioid receptor seems to be primarily responsible for these effects.[22]


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